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Treatment for non-insulin dependent diabetes melitus (NIDDM) involves the use of glibenclamide. The juvenile-onset type of diabetes, in which the pancreas has lost all or nearly all of its capacity to secrete insulin, as well as patients who have had complete pancreatectomies, render it ineffective. Such patients require insulin, and efforts to control them with oral therapy are risky and doomed to failure.


Change over from insulin to Glibenclamide: The mildly diabetic patient whose insulin requirement is fewer than 20 units daily, can be started on the initial dosage of Glibenclamide with immediate discontinuation of insulin. Daily dose of ½ tablet (2.5 mg) may be continued as maintenance therapy if good control has been achieved. For daily doses over 10 mg, two divided doses may be used. If the need for insulin is moderate or high, the switch should be made gradually by administering both insulin and glibenclamide at the same time and gradually lowering the insulin dose. If the outcome is poor, increasing the daily dose by increments of 12 tablet (2.5 mg) every 3-5 days, up to a maximum of 3 tablets, is required. Initially stabilization dosage: Glibenclamide half tablet (2.5 mg) should be taken initially during or immediately after breakfast. 3-5 days after initiation of the drug, the blood sugar level and urine sugar level should be checked. When switching from other oral anti-diabetics with a similar mechanism of action, patients should be given 1/2 to 1 tablet of glibenclamide. Only NIDDM with relatively recent onset that is being managed with low doses of insulin will transition from insulin to glibenclamide. This should preferably be done in hospital or with daily medical supervision. A patient needs insulin to get through a critical situation when their insulin needs are increased, such as during a fever, surgery, or trauma. Glibenclamide alone is insufficient in these situations.


Must be kept dry and below 30 degrees Celsius.

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Side effects

Leukopenia, thrombocytopenia, and other hemopoietic reactions caused by allergies are occasionally seen. A few possible side effects are headache, dizziness, nausea, vomiting, epigastric pain, and weakness. Glibenclamide is well tolerated.


NIDDM is treated with glibenclamide. It is ineffective in completely pancreatectomized patients and in juvenile-onset type of diabetes, in which the pancreas has lost all or nearly all of its capacity to secrete insulin. Such patients need insulin, and attempts to control them with oral therapy are risky and doomed to failure.


Regardless of the drug selected, a concerted effort must be made by the patient and the doctor to reduce the patient's weight as a necessary component of diabetic treatment. The most crucial aspect of diabetes treatment is weight loss.

Which is better glibenclamide or metformin?

Conclusions At short term, in women with gestational diabetes requiring drug treatment, glibenclamide is clearly inferior to both insulin and metformin, while metformin (plus insulin when required) performs slightly better than insulin.

Which generation is glibenclamide?

The second generation sulfonylureas include glyburide (also known as glibenclamide), gliclazide, glipizide, and glimepiride, which are oral hypoglycemic agents that are widely used in therapy of type 2 diabetes.

Why glibenclamide is used in type 2 diabetes?

Glibenclamide works by increasing the amount of insulin that your pancreas produces. This helps to reduce the amount of sugar in your blood.

Is glibenclamide better than metformin?

Treatment failure was higher with metformin than with glibenclamide. Conclusions At short term, in women with gestational diabetes requiring drug treatment, glibenclamide is clearly inferior to both insulin and metformin, while metformin (plus insulin when required) performs slightly better than insulin.

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Rachit Jain Mar 10, 2016, 6:36:51 PM

Cardiac toxicity of tesaglitazar is related mitochondrial toxicity caused by decrease in PPARγ coactivator 1-α (PPARGC1A, PGC1α) and sirtuin 1 (SIRT1). Retrieved from "" Categories: Drugs not assigned an ATC code Abandoned drugs Carboxylic acids Phenol ethers PPAR agonists Benzosulfones Gastrointestinal system drug stubs Hidden categories: All articles with dead external links Articles with dead external links from October 2010 Drugs with non-standard legal status Articles with changed ChemSpider identifier Articles with changed EBI identifier ECHA InfoCard ID from Wikidata Articles with changed InChI identifier Chemical pages without DrugBank identifier Drugboxes which contain changes to verified fields Drugboxes which contain changes to watched fields All stub articles The drug had completed several phase III clinical trials, however in May, 2006 AstraZeneca announced that it had discontinued further development.

Terry Flannery Mar 10, 2011, 10:26:56 AM

Diabetic retinopathy (DR) is a complication of diabetes and a leading cause of blindness. At the early stages of DR development, symptoms are not detectable. In patients with T2D, nerve degeneration in the retina and nerve fiber loss are linked to progressive thinning of the retina.

psteelk Aug 26, 2020, 12:06:18 PM

Tolrestat (INN) (AY-27773) is an aldose reductase inhibitor which was approved for the control of certain diabetic complications. While it was approved for marketed in several countries, it failed a Phase III trial in the U.S. due to toxicity and never received FDA approval. It was discontinued by Wyeth in 1997 because of the risk of severe liver toxicity and death.

Abel Borges Dec 17, 2015, 8:59:02 PM

Vogilbose was discovered in 1981, and was first launched in Japan in 1994, under the trade name BASEN, to improve postprandial hyperglycemia in diabetes mellitus. Voglibose (INN and USAN, trade name Voglib, marketed by Mascot Health Series) is an alpha-glucosidase inhibitor used for lowering postprandial blood glucose levels in people with diabetes mellitus. There are three major drugs which belong to this class, acarbose, miglitol and voglibose, of which voglibose is the newest. Alpha glucosidase inhibitors delay glucose absorption at the intestine level and thereby prevent sudden surge of glucose after a meal. Voglibose is a research product of Takeda Pharmaceutical Company, Japan's largest pharmaceutical company.

user7311356 May 27, 2016, 6:03:57 AM

Its effect will depend on the amount of non-monosaccharide carbohydrates in a person's diet. It must be taken at the start of main meals to have maximal effect. In contrast to acarbose (another alpha-glucosidase inhibitor), miglitol is systemically absorbed; however, it is not metabolized and is excreted by the kidneys. Miglitol is an oral anti-diabetic drug that acts by inhibiting the ability of the patient to break down complex carbohydrates into glucose.

Deepak Kumar Upadhyay Aug 21, 2020, 7:55:31 PM

As shown in the literature and in the clinical studies performed with AMGLIDIA, the average daily dose is expected to be around 0.2 to 0.5 mg/kg/day in most of the patients suffering from neonatal diabetes. From week 2 onward, if capillary blood glucose is ≥7 mmol/L increase AMGLIDIA by 0.2 mg/kg/day and reduce insulin. The bottle does not need to be shaken before administration. Patients can be discharged when no longer requiring insulin treatment, when stable on a combination of AMGLIDIA and insulin or when stable on insulin alone.

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